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dc.contributor.authorAlexander, George Lionelen
dc.date.accessioned2019-02-15T14:32:20Z
dc.date.available2019-02-15T14:32:20Z
dc.date.issued1957
dc.identifier.urihttp://hdl.handle.net/1842/34885
dc.description.abstracten
dc.description.abstractIn this Thesis some original observations concerning the Sturge Weber syndrome will be placed on record, and new deductions therefrom and from the literature will be submitted. A survey of the literature reveals that many cases described as examples of the Sturge Weber syndrome can hardly be accepted as such; some are cases of intracerebral racemose angioma, some of the patients have a facial naevus but scanty neurological symptoms or deficits are recorded; a few lack the facial cutaneous stigma. A rather rigid definition of the Sturge Weber syndrome seems therefore justifiable and desirable. The Sturge Weber syndrome is characterised by cutaneous angiomatosis (capillary naevus, naevus flammeus) affecting the face, epilepsy, gyriform calcifications within the cerebral cortex in relation to an angiomatosis at leptomeningeal level, and in many cases, buphthalmos or glaucoma. The intracerebral calcifications are not visible radiologically at birth. When the lower part of the face is included in the naevoid area the soft tissues of the face, notably the lips and the buccal and alveolar mucosa, are often hypertrophied.en
dc.publisherThe University of Edinburghen
dc.relation.ispartofAnnexe Thesis Digitisation Project 2019 Block 22en
dc.relation.isreferencedbyen
dc.titleObservations on the sturge-weber syndromeen
dc.typeThesis or Dissertationen
dc.type.qualificationlevelDoctoralen
dc.type.qualificationnameMD Doctor of Medicineen


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